glutathione and triple negative breast cancer Metabolic reprogramming in triple-negative Induction of Oxidative Stress and
Induction of Oxidative Stress and Ferroptosis in Triple Negative Breast Cancer Cells by Niclosamide via Blockade of the Function and Expression of SLC38A5 and SLC7A11 Glutathione peroxidase 4 (GPX4) and obesity interact to impact tumor progression and treatment response in triple negative breast cancer Cancer & Metabolism Springer Nature Link cd44 glutathione Receptor Mediated Ferroptosis Induction by Hyaluronic Acid Carbon Quantum Dots in Triple Negative Breast Cancer Cells Through Downregulation of SLC7A11 Pathway Reduction sensitive CD44 receptor targeted hyaluronic acid CD36 enhances sensitivity of triple negative breast cancer cells to palmitate induced ferroptosis Cell Death & Disease Comprehensive single cell metabolic profiling identifies targets to sensitize triple negative breast cancer to chemo immunotherapy: Cell Reports Medicine Autophagy Related Proteins in Triple Negative Breast Cancer: From Molecular Insights to Therapeutic Applications
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